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Abstract Number: 2634
Sex differences in medication discontinuation in axial spondyloarthritis
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Abstract Number: 2635
Differences in structural lesions of the spine between patients with early axSpA and non-axSpA chronic back pain: 2-year results of the SPACE Cohort
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Abstract Number: 2636
Autoantibodies to 14-3-3 eta: A Novel Diagnostic Biomarker for Axial Spondyloarthritis
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Abstract Number: 2637
Initial Phospholipid Transferase Activity Is Predictive of Five-Year Sacroiliac Radiographic Progression in Axial Spondyloarthritis: Findings From the DESIR Cohort
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Abstract Number: 2638
ASembleNet: A Hybrid AI Model for MRI-Based Classification of Ankylosing Spondylitis
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Abstract Number: 2639
Gut Microbiome Signatures Forecast Clinical Response to Methotrexate in Treatment-Naïve Early Rheumatoid Arthritis
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Abstract Number: 2640
Effectiveness of Biological and Target-Synthetic Treatment in Patients with Rheumatoid Arthritis and Kidney Dysfunction: A Large Prospective Registry Study
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Abstract Number: 2641
Cizutamig, a BCMA T-cell engager: preclinical to clinical translation of design optimization for the treatment of autoimmune diseases
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Abstract Number: 2642
Impact of Rheumatoid Arthritis Therapeutic Classes on Risk of Major Adverse Cardiovascular Events and Venous Thromboembolism: A Population-Based Cohort Study
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Abstract Number: 2643
Microbiome Signatures in RA Treatment: Personalizing Methotrexate Therapy
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Abstract Number: 2644
Single Cell RNA-seq Profiling Reveals a Blood Monocyte Phenotype Associated with Response to TNF Inhibitor Therapy in RA Patients
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Abstract Number: 2645
Age-specific Incidence of Systemic Lupus Erythematosus in the United States: A Meta-Analysis of Data from the Centers for Disease Control and Prevention Lupus Registries
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Abstract Number: 2646
DNA methylation-based clustering reveals clinically distinct subtypes of systemic lupus erythematosus
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Abstract Number: 2647
Unlinked Paths to SLE: Divergent Associations of DNA Methylation and Polygenic Risk Scores with SLE Features
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Abstract Number: 2648
Transcriptome analysis of quiescent SLE cases uncovers dysregulated pathways associated with disease flares
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