ACR Meeting Abstracts

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Abstracts tagged "Arthritis"

  • Abstract Number: 58 • 2017 Pediatric Rheumatology Symposium

    Analysis and Implications of Non-Invasive Knee Acoustical Emissions in Juvenile Idiopathic Arthritis

    Daniel Whittingslow1,2, Beren Semiz3, Lori Ponders4, Andrew Wiens5, Omer Inan3,6 and Sampath Prahalad7, 1Emory University School of Medicine, Atlanta, GA, 2Department of Biomedical Engineering, Georgia Institute of Technology, Atlanta, GA, 3Electrical and Computer Engineering, Georgia Institute of Technology, Atlanta, GA, 4Emory University, Atlanta, GA, 5Computer Science, Cornell University, Ithaca, NY, 6Biomedical Engineering, Georgia Institute of Technology, Atlanta, GA, 7Pediatrics, Emory Children's Center, Atlanta, GA

    Background/Purpose: Juvenile Idiopathic Arthritis (JIA), the most common chronic rheumatic disease occurring in childhood, is an important cause of disability.  Despite the persistence of disease…
  • Abstract Number: 173 • 2016 ACR/ARHP Annual Meeting

    S100A8/A9, a Potent Serum and Molecular Imaging Biomarker for Synovial Inflammation and Joint Destruction in Seronegative Experimental Arthritis

    Edwin J. W. Geven1, Martijn H. J. van den Bosch1, Shahla Abdolahi-Roodsaz1, Annet W. Sloetjes1, Sven Hermann2, Michael Schäfers2, Marije I. Koenders1, Dirk Föll3, Johannes Roth4, Thomas Vogl4 and Peter L. E. M. van Lent1, 1Experimental Rheumatology, Radboud university medical center, Nijmegen, Netherlands, 2European Institute for Molecular Imaging, University of Münster, Münster, Germany, 3Department of Pediatric Rheumatology and Immunology, University of Münster, Münster, Germany, 4Institute of Immunology, University of Münster, Münster, Germany

    Background/Purpose: Seronegative joint diseases, including psoriatic arthritis and juvenile idiopathic arthritis, are characterized by the lack of autoantibodies, which are relevant biomarkers for predicting disease…
  • Abstract Number: 1450 • 2016 ACR/ARHP Annual Meeting

    Regulatory Mechanisms of Mesenchymal Stem Cell Transplantation on Systemic Osteoporosis in Collagen-Induced Arthritis Mice

    Chang Liu, Huayong Zhang, Xiaojun Tang, Ruihai Feng, Genhong Yao, Weiwei Chen, Wenchao Li and Lingyun Sun, Department of Rheumatology and Immunology, The Affiliated Drum Tower Hospital of Nanjing University Medical School, Nanjing, China

    Background/Purpose:  To investigate the effects of umbilical cord-mesenchymal stem cell (UC-MSC) transplantation on joint damage and systemic osteoporosis in collagen induced arthritis (CIA) mice. Methods:…
  • Abstract Number: 3165 • 2016 ACR/ARHP Annual Meeting

    HLA-B27 Expression Is Accompanied By a Profoundly Altered IgA Response to the Intestinal Microbiota and Microbial Translocation to the Joint

    Mark Asquith1, Sean Davin1, Patrick Stauffer1, Claire Mitchell2 and James T. Rosenbaum1, 1Oregon Health & Science University, Portland, OR, 2Division of Arthritis and Rheumatology, Oregon Health & Science University, Portland, OR

    Background/Purpose:  HLA-B27 is the strongest known genetic risk factor for ankylosing spondylitis and other spondyloarthropathies (SpAs). We have shown previously that Fisher 344 rats that…
  • Abstract Number: 459 • 2016 ACR/ARHP Annual Meeting

    Resident Non-Classical Monocytes Are Critically Important for Tissue Destruction in Arthritis

    Antonia Puchner1, Victoria Saferding2, Michael Bonelli3, Carl-Walter Steiner2, Eliana Goncalves-Alves3, Silvia Hayer4, Yohei Mikami5, Marije M. Koenders6, Birgit Niederreiter7, Josef S. Smolen8, Kurt Redlich3, Stephan Blüml9 and Michael Bonelli and Stephan blueml, 1Department of Rheumatology, Medical University of Vienna, Vienna, Austria, 2Rheumatology, Medical University of Vienna, Vienna, Austria, 3Division of Rheumatology, Medical University of Vienna, Vienna, Austria, 4Waehringer Guertel 18-20 A-A09, Medical University of Vienna, Vienna, Austria, 5Molecular Immunology and Inflammation Branch, NIAMS, Bethesda, MD, 6Rheumatology Research and Advanced Therapeutics, Radboud University Nijmegen Medical Center, Nijmegen, Netherlands, 7Rheumatology, Internal Medicine III, Medical University of Vienna, Vienna, Austria, 8Medical University of Vienna, Vienna, Austria, 9Internal Medicine 3; Division of Rheumatology, Medical University of Vienna, Vienna, Austria

    Background/Purpose: Bone destruction in rheumatoid arthritis is mediated by osteoclasts, which are derived from precursor cells of the myeloid lineage. Although there is much known…
  • Abstract Number: 1452 • 2016 ACR/ARHP Annual Meeting

      18 F-FDG PET Imaging: An In Vivo quantitative Drug Screening Tool for Novel Antiinflammatory Therapies

    Siba P. Raychaudhuri1, Smriti K. Raychaudhuri2, Anupam Mitra3 and Abhijit Chaudhari4, 1Davis, CA, 2Rheumatology/Immunology, VA Sacramento Medical Center, Davis, CA, 3Dermatology, UC Davis School of Medicine, Davis, CA, 4Radiology, UC Davis School of Medicine, Sacramento, CA

    Background/Purpose: Collagen induced arthritis (CIA) mouse model is used for screening of new drugs for autoimmune arthritis. The conventional read outs of this model are…
  • Abstract Number: 462 • 2016 ACR/ARHP Annual Meeting

    Selective Inhibition of MMP9 Using a Monoclonal Antibody As a Therapeutic Strategy for Rheumatoid Arthritis

    Sunhwa Kim1, Brian Carr1, Leah Tong1, Debi Jin1, Ruth Wang1, Derrek Marshall1, David Gossage2 and Victoria Smith1, 1Biology, Gilead Sciences, Inc., Foster city, CA, 2Biology, Gilead Sciences, Inc., foster city, CA

    Background/Purpose: Matrix metalloproteinase-9 (MMP9), highly expressed by infiltrating inflammatory cells, pannus tissue, and multinucleated cells in the synovium and subchondral bone tissue, including osteoclasts, participates…
  • Abstract Number: 1455 • 2016 ACR/ARHP Annual Meeting

    Collagen-Induced Arthritis and Uveitis in Mice Lacking TNF Receptors

    Chiharu Iwahashi1, Minoru Fujimoto2, Tomoharu Ohkawara3, Hayato Urushima1, Satoshi Serada1 and Tetsuji Naka4, 1Laboratory for Immune Signal, National Institute of Biomedical Innovation, Health and Nutrition, Ibaraki, Japan, 2Laboratry of immune signal, National Institute of Biomedical Innovation, Health and Nutrition, Ibaraki, Japan, 3Laboratory for immune signal, National Institute of Biomedical Innovation, Health and Nutrition, Osaka, Japan, 4Laboratory for immune signal, National Institute of Biomedical Innovation, Health and Nutrition, Ibaraki, Japan

    Background/Purpose: Tumor necrosis factor (TNF) is a critical effector of the autoimmune inflammation in rheumatoid arthritis (RA) and uveitis. TNF inhibitors are preferably used in…
  • Abstract Number: 466 • 2016 ACR/ARHP Annual Meeting

    Sexually Dimorphic Dysbiosis of Gut Microbiota in Tumor Necrosis Factor Transgenic Mice with Inflammatory-Erosive Arthritis

    Richard Bell1,2, Ronald Wood3, Christopher T. Ritchlin4, Edward Schwarz5 and Homaira Rahimi6, 1Center for Musculoskelatal Research, University of Rochester, Rochester, NY, 2Pathology, University of Rochester, Rochester, NY, 3University of Rochester, Rochester, NY, 4Allergy Immunology & Rheumatology, University of Rochester Medical Center, Rochester, NY, 5Orthopedeatrics, University of Rochester, Rochester, NY, 6Rheumatology, University of Rochester/Golisano Children's Hosp, Rochester, NY

    Background/Purpose: Recent studies identify gut microbiota dysbiosis as a possible contributor to rheumatoid arthritis (RA) pathogenesis. RA patients have significantly different microbiomes than healthy controls.…
  • Abstract Number: 1477 • 2016 ACR/ARHP Annual Meeting

    Exploring the Inadequate Cardiovascular Disease Prevention in Inflammatory Joint Diseases: Results from a Nationwide Norwegian Project

    Eirik Ikdahl1, Silvia Rollefstad2, Grunde Wibetoe3, Anne Salberg4, Dag Magnar Soldal5, Inge C Olsen6, Tore K Kvien7, Anne Grete Semb1 and Glenn Haugeberg8, 1Preventive Cardio-Rheuma clinic, Dept. of Rheumatology, Diakonhjemmet Hospital, Oslo, Norway, 2Preventive Cardio-Rheuma Clinic, Department of Rheumatology, Diakonhjemmet Hospital, Oslo, Norway, 3Rheumatology, Diakonhjemmet Hospital, Oslo, Norway, 4Hospital for Rheumatic Diseases, Lillehammer, Norway, 5Rheumatology, Hospital of Southern Norway, Kristiansand, Norway, 6Department of Rheumatology, Diakonhjemmet Hospital, Oslo, Norway, 7Dept. of Rheumatology, Diakonhjemmet Hospital, Oslo, Norway, 8Martina Hansens Hospital, Bærum, Norway

    Background/Purpose: Antihypertensives (antiHT) and lipid lowering therapies (LLT) prevent cardiovascular disease (CVD) effectively. It has been reported that patients with rheumatoid arthritis (RA) receive suboptimal…
  • Abstract Number: 481 • 2016 ACR/ARHP Annual Meeting

    Dual Effect of 3-Bromopyruvate on Both Th17 and Treg Cell Differentiation and Dendritic Cell Activation Ameliorates Autoimmune Arthritis in Mice

    Takaichi Okano1, Jun Saegusa2, Keisuke Nishimura3, Yo Ueda4, Sho Sendo2, Soshi Takahashi5, Kengo Akashi3, Akira Onishi6 and Akio Morinobu2, 1Rheumatology and Clinical immunology, Kobe University Graduate School of Medicine, Kobe, Japan, 2Rheumatology and Clinical Immunology, Kobe University Graduate School of Medicine, Kobe, Japan, 3Department of Rheumatology, Kobe University Hospital, Kobe, Japan, 4Kobe University Graduate School of Medicine, Kobe, Japan, 5Department of Rheumatology and Clinical Immunology, Kobe University Graduate School of Medicine, Kobe, Japan, 6Department for Rheumatology, Kobe University Hospital, Kobe, Japan

    Background/Purpose: Recent studies have shown that cellular metabolism plays an important role in regulating immune cell function. In the process of cell differentiation, both interleukin-17-producing…
  • Abstract Number: 1915 • 2016 ACR/ARHP Annual Meeting

    Partial Elimination of Intestinal Microbiota Dampens T Helper 17 Cell Differentiation and Established Collagen-Induced Arthritis in Mice

    Rebecca Rogier1, Heather Evans-Marin2, Birgitte Walgreen1, Monique M. Helsen1, Liduine van den Bersselaar1, Peter M. van der Kraan1, Fons A.J. van de Loo3, Peter L. van Lent1, Jose U. Scher4, Wim B. van den Berg1, Marije I. Koenders1 and Shahla Abdolahi-Roodsaz1, 1Experimental Rheumatology, Radboud university medical center, Nijmegen, Netherlands, 2Division of Rheumatology, New York University School of Medicine, New York, NY, 3Rheumatology, Radboud university medical center, Nijmegen, Netherlands, 4New York University School of Medicine, New York, NY

    Background/Purpose: High-throughput sequencing of intestinal microbiota recently revealed that the composition of intestinal microbiota is perturbed in patients with new onset untreated rheumatoid arthritis (RA).…
  • Abstract Number: 925 • 2016 ACR/ARHP Annual Meeting

    Complement C5a Receptor Is the Key Initiator of Neutrophil Adhesion and Inflammation in Immune Complex-Induced Arthritis

    Yoshishige Miyabe1, Chie Miyabe1, Thomas Murooka2, Edward Kim3, Nancy Kim3, Thorsten R. Mempel4 and Andrew D. Luster1, 1Rheumatology, Allergy and Immunology, Massachusetts General Hospital, Harvard Medical School, Boston, MA, 2University of Manitoba, Winnipeg, MB, Canada, 3Massachusetts General Hospital, Boston, MA, 4Massachusetts General Hospital, Charlestown, MA

    Background/Purpose: Inflammatory arthritis, including rheumatoid arthritis, is characterized by neutrophil (PMN) recruitment into the joint in a highly regulated process controlled by chemoattractants (CAs). Four…
  • Abstract Number: 2157 • 2016 ACR/ARHP Annual Meeting

    Share the Fate: Fibroblast-like Synoviocyte Cell-to-Cell Organelle Transfer Is Directed By the Inflammatory Microenvironment

    Ruth Byrne1, Isabel Olmos Calvo2, Thomas Karonitsch3, Felix Kartnig4, Johannes Holinka5, Günter Steiner6, Peter Ertl7, Josef Smolen8 and Hans Peter Kiener9, 1Rheumatology, Internal Medicine III, Medical University of Vienna, Vienna, Austria, 2Nanotechnology, Austrian Institute for Technology, Vienna, Austria, 3Internal Medicine III, Vienna Medical University, Vienna, Austria, 4Department of Medicine III, Division of Rheumatology, Medical University of Vienna, Vienna, Austria, 5Department of Orthopaedics, Medical University of Vienna, Vienna, Austria, 6Internal Medicine III, Division of Rheumatology, Medical University of Vienna, Vienna, Austria, 7Vienna University of Technology, Vienna, Austria, 8Medical University of Vienna and Hietzing Hospital, Vienna, Austria, 9Division of Rheumatology, Medical University of Vienna, Vienna, Austria

    Background/Purpose: Fibroblast-like synoviocytes (FLS) form a complex tissue network via long-distance intercellular connections with wide intercellular matrix spaces. The adaptive synovial tissue response to inflammation…
  • Abstract Number: 1008 • 2016 ACR/ARHP Annual Meeting

    Advantageous Effect of an Endogenous Retroviral Envelope Protein in Systemic Lupus Erythematosus with Ex Vivo and In Vivo Anti-Inflammatory Potential

    Anne Troldborg1,2, Magdalena Janina Laska3, Ellen-Margrethe Hauge4,5, Shervin Bahrami6 and Kristian Stengaard-Pedersen7,8, 1clinical medicine, Aarhus University, Aarhus, Denmark, 2Rheumatology, Aarhus University Hospital, Aarhus, Denmark, 3Biomedicine, Aarhus University, Aarhus, Denmark, 4Dept. of Anatomy, Aarhus University, Aarhus, Denmark, 5Rheumatology, Department of Rheumatology, Aarhus University Hospital, Aarhus, Denmark, 6Clinical Medicine, Aarhus University, Aarhus, Denmark, 7Clinical medicine, Aarhus University, Aarhus, Denmark, 8Department of Rheumatology, Aarhus University Hospital, Aarhus, Denmark

    Background/Purpose: Human Endogenous Retroviruses (HERVs) are remnants of retroviral infections in the human germline. Most, but not all, HERV genes have become inactive by accumulation…
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All abstracts accepted to ACR Convergence are under media embargo once the ACR has notified presenters of their abstract’s acceptance. They may be presented at other meetings or published as manuscripts after this time but should not be discussed in non-scholarly venues or outlets. The following embargo policies are strictly enforced by the ACR.

Accepted abstracts are made available to the public online in advance of the meeting and are published in a special online supplement of our scientific journal, Arthritis & Rheumatology. Information contained in those abstracts may not be released until the abstracts appear online. In an exception to the media embargo, academic institutions, private organizations, and companies with products whose value may be influenced by information contained in an abstract may issue a press release to coincide with the availability of an ACR abstract on the ACR website. However, the ACR continues to require that information that goes beyond that contained in the abstract (e.g., discussion of the abstract done as part of editorial news coverage) is under media embargo until 10:00 AM ET on November 14, 2024. Journalists with access to embargoed information cannot release articles or editorial news coverage before this time. Editorial news coverage is considered original articles/videos developed by employed journalists to report facts, commentary, and subject matter expert quotes in a narrative form using a variety of sources (e.g., research, announcements, press releases, events, etc.).

Violation of this policy may result in the abstract being withdrawn from the meeting and other measures deemed appropriate. Authors are responsible for notifying colleagues, institutions, communications firms, and all other stakeholders related to the development or promotion of the abstract about this policy. If you have questions about the ACR abstract embargo policy, please contact ACR abstracts staff at [email protected].

ACR Abstract Embargo Policy

Accepted abstracts are made available to the public online in advance of the meeting and are published in a special online supplement of Arthritis & Rheumatology. Information contained in those abstracts may not be released until the abstracts appear online. Academic institutions, private organizations and companies with products whose value may be influenced by information contained in an abstract may issue a press release to coincide with the availability of an ACR abstract on the ACR website. However, the ACR continues to require that information that goes beyond that contained in the abstract (e.g., discussion of the abstract done as part a scientific presentation or presentation of additional new information that will be available at the time of the meeting) is under embargo until Saturday, November 11, 2023.

Violation of this policy may result in the abstract being withdrawn from the meeting and other measures deemed appropriate. Authors are responsible for notifying financial and other sponsors about this policy. If you have questions about the abstract embargo policy, please contact the public relations department at [email protected].

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