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Abstract Number: 1005
Regulation of Neutrophil Activation in Systemic Lupus Erythematosus
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Abstract Number: 1006
Physical Activity at Lower Intensities Reduces Localized IL-1b in a Murine Model of Gout By Systemically Down-Regulating TLR2 Expression on Circulating Neutrophils and Suppressing CXCL1 Expression
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Abstract Number: 1007
Macrophage Mediators of Autoimmune Valvular Carditis and Fibrosis
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Abstract Number: 1008
Presence of a Specific Defect in M2 Polarization of Blood Monocytes from Patients with Rheumatoid Arthritis, Associated with Increased microRNA-155
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Abstract Number: 1009
Monocytes Membrane TNF Expression and Anti-TNF Treatment in Rheumatoid Arthritis
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Abstract Number: 1010
Inhibition of Bruton’s Tyrosine Kinase (BTK) Prevents Inflammatory Macrophage Differentiation: A Potential Role in RA and SLE
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Abstract Number: 1011
Tissue Resident Macrophages Establish a Niche That Limits Monocyte to Macrophage Differentiation in Synovial Tissue during Homeostasis
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Abstract Number: 1012
Inhibition of Inflammation and Oxidative Stress in Systemic Sclerosis (SSc) Macrophages
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Abstract Number: 1013
Circulating Hybrid M1/M2 Monocytes/Macrophages in Systemic Sclerosis Patients with Lung Involvement
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Abstract Number: 1014
Distinct Macrophage Phenotype and Bioenergetic Profiles in Rheumatoid Arthritis
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Abstract Number: 1015
Rheumatoid Arthritis Peripheral CD14+ Monocytes Are Hyper-Inflammatory, Hyper-Glycolytic and Retain a Memory Bias Toward M1 Macrophages
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Abstract Number: 1016
The Distinct Profile of Ly6clo Monocytes in the Murine Joint Compared with Those in Circulation Suggests a Unique Role in Inflammatory Arthritis
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Abstract Number: 1017
Autoantigen Pentraxin-3 Is an Inflammatory Cytokine Storms Suppressor By Switching Monocytes Pyroptosis to Apoptosis in a Complement-Dependent Manner in Rheumatoid Arthritis
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Abstract Number: 1018
Tofacitinib Impairs Monocyte-Derived Dendritic Cell Differentiation in Rheumatoid Arthritis and Psoriatic Arthritis.
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Abstract Number: 1019
Involvement of Mast Cells in the Pathogenesis of Sjögren’s Syndrome By Induction of Tissue Fibrosis Via Fibroblast Collagen Synthesis
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