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Home » Meeting Abstracts » ACR/ARHP Annual Meeting 2017

Abstract Number: 2346

Chemokine Ligand 9 (CXCL9) [Monokine Induced By Gamma Interferon (MIG)]  As a Predictor of Active Disease Status in Localized Scleroderma

Kathryn S. Torok1, Qi Mi2, Emily Mirizio3, Kaila Schollaert-Fitch1, Mark Fritzler4 and Marvin J. Fritzler5, 1Pediatric Rheumatology, University of Pittsburgh Med Ctr, Pittsburgh, PA, 2Department of Sports Medicine and Nutrition, University of Pittsburgh, Pittsburgh, PA, 3Pediatric Rhuematology, University of Pittsburgh Med Ctr, Pittsburgh, PA, 4Eve Technologies, Calgary, AB, Canada, 5Medicine, University of Calgary, Calgary, AB, Canada

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Meeting: ACR/ARHP Annual Meeting 2017

Date of first publication: September 18, 2017

Keywords: cytokines, Disease Activity, Morphea, pediatric rheumatology and scleroderma

Session Information

Date: Tuesday, November 7, 2017

Title: Pediatric Rheumatology – Pathogenesis and Genetics Poster

Session Type: ACR Poster Session C

Session Time: 9:00AM-11:00AM

Background/Purpose:

Localized scleroderma (LS) is a fibrotic autoimmune disease of the skin and underlying tissues which can lead to disfiguring sequlea, especially in childhood-onset.  Untreated active disease is associated with long-term damage and disability; therefore accurate assessment of the active disease state is imperative, and a distinct activity biomarker would assist clinicians to intervene before disease progression. The purpose of this study was to investigate a wide variety of cyto/chemokines, specifically within disease transition from active to inactive phases in individual patients, which correlate to established clinical measures of disease activity in LS.

Methods:

Plasma from 70 juvenile LS (jLS) patients with clinical data and longitudinal samples (average of 6 years follow-up), including at least one active and one inactive specimen (272 samples total), were evaluated by microarray to study a 60-analyte cyto/chemokine panel. This included the study of TH1, TH2, and TH17 associated cytokines with an additional IFN-γ panel that emphasized CXCL9, CXCL11, and MIP-3β chemokines. Wilcoxon Signed-Ranks Test (p<0.05) was used to compare cyto/chemokines between active and inactive disease subsets. Next, to focus on clinically significant cytokines, additional analyses included spearman’s correlation of analytes with the modified Localized Skin Severity Index (mLoSSI), a validated active disease variable, followed by multiple regression analyses of the significantly correlated cytokines using R language.

Results:

When dichotomizing the LS samples into active vs. inactive subjects, many reoccurring cytokines were significantly elevated (Table 1 – left column). Correlation of the 60 analyte panel resulted in moderate to strong correlation of IFN-γ and TH1-like associated cyto/chemokines with the mLoSSI, including CXCL9 (MIG), MIP-3β, and IL-10 (Table 1 – right column). Of those that correlated strongly with the mLoSSI, IL-10 and CXCL9 were statistically significant predictors in multiple linear regression analyses (p = 0.002 and 0.001, respectively), with an adjusted R-squared of 0.71; meaning the change of IL-10 and CXCL9 are related to the change in mLoSSI, signifying change in disease activity status.

Conclusion:

This is a unique examination of circulating cytokines in jLS and further correlates the biological activity to clinical disease activity measures used for disease monitoring, the mLoSSI. The correlated chemokines indicate a strong IFN-γ associated environment. CXCL9 is a predominant IFN-γ inducible chemokine and is part of the IFN-γ family that contributes to the activation of the M1 phenotype and recruitment of TH1 cells, both of which induce further inflammatory cytokine response. Further analysis using the best-fit model is underway with the overall goal of finding the strongest core set of biomarkers that highly predicts the mLoSSI in LS patients, signifying degree of disease activity.

 

Table 1: Analyses of 60 cyto/chemokines in LS with focus on disease activity

 

Active vs. Inactive LS peripheral blood cyto/chemokine

 

Correlation of the mLoSSI with the peripheral blood cyto/chemokines

 

Cyto/chemokine

 

 

Cyto/chemokine

 

 

 

 

p-value

 

 

Spearman’s correlation

p-value

 

CXCL9 (MIG)

0.030

 

IL-10

0.46

0.0005

 

CXCL11 ( I-TAC)

0.050

 

CXCL9 (MIG)

0.42

0.0018

 

MIP-3β

0.001

 

CXCL10 (IP-10)

0.57

0.0112

 

IL-9

0.020

 

GM-CSF

0.38

0.0049

 

IL-2

0.020

 

IFN-γ

0.33

0.0161

 

CCL-1 (-309)

0.020

 

MIP-3β

0.30

0.0255

 

 

 

TNF-α

0.30

0.0266

 

 

 


Disclosure: K. S. Torok, None; Q. Mi, None; E. Mirizio, None; K. Schollaert-Fitch, None; M. Fritzler, Eve Technologies, 3; M. J. Fritzler, Inova Diagnostics, Inc., 5.

To cite this abstract in AMA style:

Torok KS, Mi Q, Mirizio E, Schollaert-Fitch K, Fritzler M, Fritzler MJ. Chemokine Ligand 9 (CXCL9) [Monokine Induced By Gamma Interferon (MIG)]  As a Predictor of Active Disease Status in Localized Scleroderma [abstract]. Arthritis Rheumatol. 2017; 69 (suppl 10). https://acrabstracts.org/abstract/chemokine-ligand-9-cxcl9-monokine-induced-by-gamma-interferon-mig-as-a-predictor-of-active-disease-status-in-localized-scleroderma/. Accessed .

ACR Meeting Abstracts - https://acrabstracts.org/abstract/chemokine-ligand-9-cxcl9-monokine-induced-by-gamma-interferon-mig-as-a-predictor-of-active-disease-status-in-localized-scleroderma/

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