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Abstract Number: 2531
CRISPR Deletion Screen in Fibroblasts Identifies Novel Regulators of Inflammation
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Abstract Number: 2532
Mortality in Patients with Pre-existing Autoimmune Disease on Immune Checkpoint Inhibitor Therapy
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Abstract Number: 2533
Genome-wide Association of Rheumatoid Arthritis in the African Ancestry Identifies HLA Amino Acid Polymorphisms of Risk
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Abstract Number: 2534
Deciphering Complement-dependent Macrophage Phenotypes in Human Rheumatoid Arthritis Using Combined Computational-experimental Single-cell Omics
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Abstract Number: 2535
The Lipidomic and Proteomic Profiles in Antiphospholipid Syndrome Patients Are Intricately Linked to Disease Pathogenesis and Modulated by Ubiquinol Supplementation
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Abstract Number: 2536
Trans-Disease Microbial Biomarkers of Protection and Pathogenesis in Autoimmune Conditions: Results from the AMP AIM Consortium
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Abstract Number: 2537
Sjögren’s Disease and Non-Sjögren’s Sicca Patient Subsets Exhibit Cell Type-specific Transcriptional Dysregulations That May Identify Early Molecular Predictors of Disease Transition
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Abstract Number: 2538
HLA-DRB1 Rheumatoid Arthritis (RA) Risk Alleles Preferentially Select TRBJ2-3-containing CD4 T Cells in RA Patients
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Abstract Number: 2539
Telocytes Integrated into Mast Cells and Joint-Draining Lymphatic Vessels Potentially Regulate Lymphatic Clearance
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Abstract Number: 2540
Characterizing the Functional Role of Type I Interferons in Inflammatory Arthritis
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Abstract Number: 2541
Probiotic Modulation of Gut Microbiota Mitigates Early Rheumatoid Arthritis Progression: Insights from Pre-Clinical Models
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Abstract Number: 2542
Group 2 Innate Lymphoid Cells Aggravates Development of Inflammatory Arthritis
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Abstract Number: 2543
Novel IgG Degrader BHV-1300 Demonstrates the Ability to Remove Anti-bDMARD ADA and Allows for Co-administration with Fc-containing Biologics
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Abstract Number: 2544
Spatial Transcriptomics Suggests Synovial Macrophage Niches Are Sexually Dimorphic in a Mouse Inflammatory Arthritis Model
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Abstract Number: 2545
Phosphodiesterase 1B Contributes to Neuropsychiatric Manifestations in Lupus-Prone Mice Through Microglial Activation
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