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Abstract Number: 1826
Unveiling the Primary Cilia Signature Driving Systemic Sclerosis Pathogenesis
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Abstract Number: 1827
Characterization of Fibroblast Subpopulations and Their Cellular Local Environments in Systemic Sclerosis Using Imaging Mass Cytometry
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Abstract Number: 1828
SSc Fibroblasts Trigger a Systemic Type I Interferon Response in SSc Patients Through Canonical TGF-β Receptor Signalling
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Abstract Number: 1829
Genomic Analysis of Skin Biopsies Differentiates Major Anti-Nuclear Autoantibody Subsets in Limited Cutaneous Systemic Sclerosis
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Abstract Number: 1830
Single-cell RNA Sequencing Profiling of Very Early-Stage Systemic Sclerosis Skin Reveals a Fibroblast Pro-inflammatory Gene Signature and Keratinocyte Dysregulation
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Abstract Number: 1831
Role of Long Non-Coding RNA H19 in Mediating Skin Fibrosis in Systemic Sclerosis
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Abstract Number: 1832
Targeting Hippo Pathway Diminishes Disease Signaling in Scleroderma Skin
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Abstract Number: 1833
Immune Complexes of Antibodies Directed to Angiotensin Receptor type-1 and Extracellular Vesicles Are Unique Modulators Contributing to Systemic Sclerosis
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Abstract Number: 1834
Altered Mechanotransduction via Myosin II Contributes to Collagen and IL-6 Production in Systemic Sclerosis Skin
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Abstract Number: 1835
FoxP3Hi CTLA4+ ICOS+ Regulatory T Cells Are Expanded in Patients with Sarcoidosis but Not Systemic Sclerosis or IgG4-Related Disease
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Abstract Number: 1836
Transcriptional Changes in the Formation of Tissue Resident Memory T Cells in the Joint
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Abstract Number: 1837
Baseline Multiome Sequencing of CD45RO+CD45RA-CD4+ T Cell Reveals Distinct Immune Profiles Associated with Subsequent Response to Secukinumab Treatment
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Abstract Number: 1838
M5542: A Potent CD80, CD86, and OX40L Antagonist Fusion Molecule for the Treatment of Autoimmune Diseases
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Abstract Number: 1839
PPP2R3C Overexpression Suppresses TCR -mediated CD4+ T Cell Abnormal Activation in Systemic Lupus Erythematosus via JNK and AKT-mTOR Pathways
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Abstract Number: 1840
Synovial Resident Memory T Cell Formation During Inflammation Requires Cell Contact with Fibroblast-Like Synoviocytes
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