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Abstract Number: 163
Fibromyalgia Patients Identify More Causes of Disease Flare Ups Than RA Patients
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Abstract Number: 164
Association of Natural Killer Cell Ligand Polymorphism, HLA-C Asn80Lys, with the Development of Anti-SSA/Ro Associated Congenital Heart Block
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Abstract Number: 165
Rheumatoid Arthritis Risk Polymorphisms in CCR6, SNP and Estrogen-Dependent Response to Immune Mediator Gene Expression, and NF-κb Transcriptional Activity: Crosstalk between the Immune and Endocrine Systems
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Abstract Number: 166
Integrative Systems Biology Approach Identifies Key Transcription Factors and Novel Rheumatoid Arthritis (RA) and Individualized Therapeutic Targets
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Abstract Number: 167
Differentially Co-Expressed Gene Networks in Previously DMARD-Naïve Patients with Early RA Achieving Sustained Drug-Free Remission after Step-up Methotrexate Therapy
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Abstract Number: 168
Identification of Novel Susceptibility Loci in a Large UK Cohort of Juvenile Idiopathic Arthritis (JIA) Cases
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Abstract Number: 169
New Autoinflammatory Phenotype Associated with Homozygous AGBL3 Variant
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Abstract Number: 170
Genome-Wide Association Study of Clinically-Ascertained Gout and Subtypes Identifies Multiple Susceptibility Loci Including Transporter Genes
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Abstract Number: 171
Finding Transcriptional Regulators Central to RA with Transcriptomics of IL17 Dose Response, Time Series, and siRNA Silencing in Stromal Cells
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Abstract Number: 172
Optimizing Precision Medicine By Using Genetics to Assign Diagnostic Prior Probabilities to Patients with Synovitis
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Abstract Number: 173
Applying Urine Proteomics for Discovery of Lupus Nephritis Damage Biomarkers in a Pediatric Cohort
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Abstract Number: 174
Transcriptional Profiling of Synovial Macrophages from RA Patients to Capture Disease Heterogeneity
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Abstract Number: 175
Precisely Controlled Differential Gene Expression System to Investigate the Effect of eQTL
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Abstract Number: 176
SEC16A and Antigen Presentation Abnormalities in the Pathogenesis of Axial Spondyloarthritis
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Abstract Number: 177
Intronic Variants of the B-Cell Proliferator RASGRP3 Affect Its Expression, and Might Contribute to Lupus Risk
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