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Abstract Number: 1902
Detecting the Pulmonary Vascular Involvement and the Changes in Gene Activation Profiles at Early Stage of Systemic Sclerosis in Patients with Raynaud Phenomenon
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Abstract Number: 1903
Determining Disease Course in Localized Scleroderma: A Prospective Cohort Study
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Abstract Number: 1904
Elevated GITRL Is Associated with Multi-Organ Involvement and Increased Disease Activity of Primarty Sjogren’s Syndrome and Promotes Pathogenic Th1/17 Differentiation
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Abstract Number: 1905
Therapeutic Targeting of JAK/STAT Pathway Inhibits Follicular Helper T Cell Maturation and Function
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Abstract Number: 1906
Impact of Environmental Factors and Inflammation on Alterations of the Total Peripheral T-Cell Compartment in Granulomatosis with Polyangiitis
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Abstract Number: 1907
T-Bet Regulates Ahr-Mediated Th-17 Differentiation Independently of IFNγ
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Abstract Number: 1908
Identification of a Novel Pro-Inflammatory T Cell Epitope from His-tRNA-Synthetase Associated with Interstitial Lung Disease in Anti-Jo-1 Positive Patients
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Abstract Number: 1909
The Transcription Factor Fra2 Is Playing a Key Role in Treg Development and Autoimmunity
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Abstract Number: 1910
IL-21 Inhibits Treg Differentiation and Function in SLE By Modulating GATA-3 and CTLA-4
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Abstract Number: 1911
Potent and Selective Tyk2 Inhibitor Highly Efficacious in Rodent Models of Inflammatory Bowel Disease and Psoriasis
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Abstract Number: 1912
A New Avenue of Immune Regulation Conferred By Self-Glycerophospholipids Via Mobilization and Migration of Myeloid-Derived Suppressor Cells
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Abstract Number: 1913
B Cell Depletion Therapy Impact CD8 T Cells in ANCA-Associated Vasculitis
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Abstract Number: 1914
Immune Recognition of a Novel Citrullinated Epitope of Cartilage Proteoglycan Aggrecan in Mice with Proteoglycan-Induced Arthritis and in Patients with Rheumatoid Arthritis
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Abstract Number: 1915
Partial Elimination of Intestinal Microbiota Dampens T Helper 17 Cell Differentiation and Established Collagen-Induced Arthritis in Mice
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Abstract Number: 1916
Heightened MAIT Cell Sensitivity to MR1 Ligands Could Impact Control of Dysbiosis in Patients with Rheumatoid Arthritis and Ankylosing Spondylitis
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