- Abstract Number: 1052
Identification of Novel Sjogren’s Syndrome Risk Loci in the Regions of TNFAIP3 and PRDM1
- Abstract Number: 2797
Identification of Serum Biomarker for the Glandular Dysfunction of Primary Sjögren’s Syndrome
- Abstract Number: 1022
Identification of Shared Genomic Regions in Distantly Related Pairs of Cases with Juvenile Idiopathic Arthritis
- Abstract Number: 844
Identification of Subclinical Skin Involvement By High Frequency Ultrasound in Systemic Sclerosis Patients
- Abstract Number: 2136
Identification of Synovial Fibroblast Subsets That Define Pathology in Rheumatoid Arthritis
- Abstract Number: 2461
Identification of Target Antigens for Anti-Endothelial Cell Antibodies in Patients with Pediatric Rheumatic Diseases Using Proteomics
- Abstract Number: 1243
Identification of the Gene Expression Signatures Predicting the Responses to Three Biologics (infliximab, tocilizumab, and abatacept) in Rheumatoid Arthritis
- Abstract Number: 1857
Identification of the Long Noncoding RNA NEAT1 As a Novel Inflammatory Regulator Acting through MAPK Pathway in Human Lupus
- Abstract Number: 2628
Identifying Anxiety and Depression Among Rheumatoid Arthritis Patients Using the Multidimensional Health Assessment Questionnaire
- Abstract Number: 140
Identifying Psoriatic Arthritis and Ankylosing Spondylitis Patients Responsible for the Highest Costs of Care: Data from a Large US Cohort
- Abstract Number: 1583
Identifying Risk Factors for Progression of Rheumatoid Arthritis-Associated Interstitial Lung Disease
- Abstract Number: 118
Identifying the Investment Needed to Generate a Durable Prednisone Dose Decrement
- Abstract Number: 1846
IGFBP-2 As a Novel Biomarker for Disease Activity and Renal Pathology in Lupus Nephritis
- Abstract Number: 1799
IgG Levels Correlate Inversely with Proteinuria Among Participants in the Abatacept and Cyclophosphamide Combination Therapy for Lupus Nephritis Trial, but Hypogammaglobulinemia Was Not Associated with an Increased Risk of Serious Infection
- Abstract Number: 742
IgM Antibodies Against Malondialdehyde and Phosphorylcholine Are T Cell Dependent and Strong Protection Markers for Atherosclerosis in SLE
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