- Abstract Number: 1373
Epigenetic Mechanisms Contribute to the Lack of LPS-Induced Tolerance in Rheumatoid Arthritis Synovial Fibroblasts
- Abstract Number: 1624
Epigenetic Profiling of CD14+ and CD16+ Monocyte Subtypes in Rheumatoid Arthritis. Alterations Related to Cardiovascular Disease and Endothelial Dysfunction
- Abstract Number: 2466
Epigenetic Traits Correlate with Clinical Activity in Juvenile Idiopathic Arthritis
- Abstract Number: 1262
Epigenome-Wide DNA Methylation Patterns Associated with Fatigue in Primary Sjögren’s Syndrome
- Abstract Number: 3166
Epistatic Interaction of Functional Inflammasome Genetic Variants in Determining the Risk of Gout
- Abstract Number: 1683
Epithelial Changes in Response to Tocilizumab Combined with Methotrexate in Rheumatoid Arthritis: Evaluated on Circulating Fragments of Type IV Collagen
- Abstract Number: 1108
Epratuzumab, a Monoclonal Antibody Targeting CD22 on B Cells, Stimulates the Phosphorylation of Upstream Inhibitory Signals of the B Cell Receptor
- Abstract Number: 3130
Epstein-Barr Virus Induces Activation of Infammatory Markers Via the TLR8 Transduction Pathway in Infected Scleroderma Monocytes
- Abstract Number: 983
ERAP1 Deficiency Protects HLA-B27 Transgenic Rats from Arthritis
- Abstract Number: 482
Erosions in the Foot at Baseline Are Predictive of Orthopedic Shoe Use after 10 Years of Treat to Target Therapy
- Abstract Number: 2898
Erythrocytes, Acanthocytes, and Proteins in Urine Reflect Lupus Nephritis Histology
- Abstract Number: 1807
Estimated Sodium and Potassium Intake Are Associated with Blood Pressure in Patients with Systemic Lupus Erythematosus
- Abstract Number: 767
Estimating the Pre-Test Probability of Systemic Lupus Erythematosus By Utilizing Anti-Double Stranded DNA and Cell Bound Complement Activation Products Testing from Rheumatology Based Practices in the United States
- Abstract Number: 1228
Estimating Under-Diagnosis of Rheumatoid Arthritis in Primary Care Data from the UK Clinical Practice Research Datalink
- Abstract Number: 3010
Estrogens Inhibit the Profibrotic Effects of Transforming Growth Factor-Beta and Protect from the Development of Experimental Dermal Fibrosis
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