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Abstracts tagged "giant cell arteritis"

  • Abstract Number: 2360 • 2012 ACR/ARHP Annual Meeting

    Prevalence, Management and Outcomes of PET Positive Large Vessel Vasculitis in Difficult to Treat PMR and GCA Patients

    Pravin Patil1, Shaifali Jain2, Katerina Achilleos3, Tochukwu Adizie1, Mark Williams1, Matthew Tam2 and Bhaskar Dasgupta4, 1Rheumatology, Southend University Hospital, Westcliff on sea, United Kingdom, 2Radiology, Southend University Hospital, Westcliff on sea, United Kingdom, 3Rheumatology, Southend University Hospital, Westcliff-on-sea, United Kingdom, 4Rheumatology, Southend University Hospital, Westcliff-on-Sea, United Kingdom

    Background/Purpose:  Management of PMR and GCA can be challenging in patients with persistently elevated inflammatory markers, prominent constitutional symptoms and inadequate steroid response. We undertook…
  • Abstract Number: 859 • 2012 ACR/ARHP Annual Meeting

    Endothelin-1 (ET-1) Induces Extracellular Matrix Protein Production by Human Temporal Artery Derived Myointimal Cells. A Mechanism Potentially Leading to Intimal Hyperplasia and Vascular Occlusion in Giant-Cell Arteritis

    Ester Planas-Rigol1, Marc Corbera-Bellalta2, Marco A. Alba3, Itziar Tabera-Bahillo3, Sergio Prieto-Gonzalez3, Georgina espigol-Frigole3, Jose Hernandez-Rodriguez4, Ester Lozano5 and Maria C. Cid6, 1Systemic Autoimmune Diseases, Vasculitis Research Unit. Hospital Clínic. University of Barcelona. IDIBAPS, Barcelona, Spain, 2Hospital Clinic University Barcelona, Hospital Clinic University Barcelona, Barcelona, Spain, 3Vasculitis research Unit, systemic autoimmune diseases department. Idibaps., Hospital Clinic University Barcelona, Barcelona, Spain, 4Vasculitis research Unit, systemic autoimmune diseases department. Idibaps., Hospital Clínic. University of Barcelona. IDIBAPS, Barcelona, Spain, 5Vasculitis research Unit, systemic autoimmune diseases department. Idibaps., Hospital Clinic University Barcelona, 6Vasculitis Research Unit, Department of Autoimmune Diseases, Hospital Clínic University of Barcelona, Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain

    Background/Purpose: Endothelin-1 (ET-1) is the main isoform of the Endothelin family. It is also the most powerful vasoconstrictor identified. ET-1 is constitutively produced in blood…
  • Abstract Number: 2568 • 2012 ACR/ARHP Annual Meeting

    The Risk of Pulmonary Embolism and Deep Vein Thrombosis in Giant Cell Arteritis: A Population-Based Cohort Study

    J. Antonio Avina-Zubieta1, Diane Lacaille2, Eric C. Sayre3, Jacek A. Kopec3 and Hyon Choi4, 1Rheumatology, Arthritis Research Centre of Canada, University of British Columbia, Richmond, BC, Canada, 2Rheumatology, Arthritis Research Centre of Canada/University of British Columbia, Richmond, BC, Canada, 3Arthritis Research Centre of Canada, Richmond, BC, Canada, 4Section of Rheumatology and the Clinical Epidemiology Unit, Boston University School of Medicine, Arthritis Research Centre of Canada/University of British Columbia, Boston, MA

    Background/Purpose: A recent hospital-based study has suggested an 8 fold increased risk of pulmonary embolism (PE) in individuals with polymyalgia rheumatica in the year following…
  • Abstract Number: 2390 • 2012 ACR/ARHP Annual Meeting

    Color Doppler Ultrasonography an Alternative to CT/MR Angiography for Identifying Large Vessel Involvement in Giant Cell Arteritis?

    Andreas P. Diamantopoulos1, Glenn Haugeberg1 and Geirmund Myklebust2, 1Rheumatology, Hospital of Southern Norway Trust, Kristiansand, Norway, 2Rheumatology, Hospital of Southern Norway, Kristiansand, Norway

    Background/Purpose: Large vessel involvement has been reported to be present in 20-50% of patients with giant cell arteritis (GCA). Computed tomography (CT) and magnetic resonance…
  • Abstract Number: 2350 • 2012 ACR/ARHP Annual Meeting

    Similarities Exceeds Differences in the Pattern of Joint and Vascular Positron Emission/Computed Tomography Uptake in Polymyalgia Rheumatica and Giant Cell Arteritis

    Dario Camellino1, Silvia Morbelli2, Francesco Paparo3, Michela Massollo2, Gianmario Sambuceti4 and Marco A. Cimmino5, 1Dipartimento Medicina Interna, Clinica Reumatologica, Genova, Italy, 2Dipartimento Medicina Interna, Medicina Nucleare, Genova, Italy, 3Radiology Unit, E.O. Ospedali Galliera, Genoa, Italy, 4Dipartimento di Medicina Interna, Medicina Nucleare, Genova, Italy, 5Department of Internal Medicine, Academic Unit of Clinical Rheumatology, University of Genova, Genova, Italy

    Background/Purpose: Polymyalgia rheumatica (PMR) and giant cell arteritis (GCA) are two frequent overlapping diseases. The purpose of this work is to examine the relationship between…
  • Abstract Number: 2353 • 2012 ACR/ARHP Annual Meeting

    TLR2 Activation by Acute Serum Amyloid A Induces Pro-Inflammatory Mechanisms in a Novel Ex Vivo Temporal Artery Explant Culture/Model of Giant Cell Arteritis

    Peadar Rooney1, Danielle Molloy1, Jennifer McCormick2, Mary Connolly1, Sinead M. Miggin3, Ashwini Maratha3, Douglas J. Veale4, Conor Murphy5, Eamonn Molloy6 and Ursula Fearon7, 1Rheumatology, Dublin Academic Medical Centre, Dublin, Ireland, 2Dublin Academic Medical Centre, Translational Rheumatology Research Group, Dublin, Ireland, 3Biology, Immune Signalling Laboratory, Maynooth, Ireland, 4Rheumatology, St. Vincent's University Hospital, Dublin 4, Ireland, 5Department of Ophthalmology, Royal Victoria Eye and Ear Hospital, Dublin, Ireland, 6Rheumatology, Dublin Academic Medical Centre, St. Vincent's University Hospital, Dublin, Ireland, 7Rheumatology, Translation Research Group, Dublin Academic Medical Centre, St. Vincent's University Hospital, Dublin, Ireland

    Background/Purpose: Giant cell arteritis (GCA) is the most common form of primary vasculitis. The pathogenesis of this disease is characterised by disregulated angiogenesis and inflammatory…
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All abstracts accepted to ACR Convergence are under media embargo once the ACR has notified presenters of their abstract’s acceptance. They may be presented at other meetings or published as manuscripts after this time but should not be discussed in non-scholarly venues or outlets. The following embargo policies are strictly enforced by the ACR.

Accepted abstracts are made available to the public online in advance of the meeting and are published in a special online supplement of our scientific journal, Arthritis & Rheumatology. Information contained in those abstracts may not be released until the abstracts appear online. In an exception to the media embargo, academic institutions, private organizations, and companies with products whose value may be influenced by information contained in an abstract may issue a press release to coincide with the availability of an ACR abstract on the ACR website. However, the ACR continues to require that information that goes beyond that contained in the abstract (e.g., discussion of the abstract done as part of editorial news coverage) is under media embargo until 10:00 AM CT on October 25. Journalists with access to embargoed information cannot release articles or editorial news coverage before this time. Editorial news coverage is considered original articles/videos developed by employed journalists to report facts, commentary, and subject matter expert quotes in a narrative form using a variety of sources (e.g., research, announcements, press releases, events, etc.).

Violation of this policy may result in the abstract being withdrawn from the meeting and other measures deemed appropriate. Authors are responsible for notifying colleagues, institutions, communications firms, and all other stakeholders related to the development or promotion of the abstract about this policy. If you have questions about the ACR abstract embargo policy, please contact ACR abstracts staff at [email protected].

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