ACR Meeting Abstracts

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Abstracts tagged "animal models and synovial cells"

  • Abstract Number: 2546 • 2015 ACR/ARHP Annual Meeting

    Targeting Synovial Fibroblasts By the Intra-Articular Delivery of microRNA-140-3p and -5p Ameliorates Experimental Autoimmune Arthritis

    Chrong-Reen Wang1, Jia-Shiou Peng2, Shih-Yao Chen3, Chao-Liang Wu4 and Ai-Li Shiau5, 1Rheum/Immun Sec/Int Med Dept, Medical Coll/Nat'l Cheng Kung, Tainan, Taiwan, 2Institute of Basic Medical Sciences, Medical Coll/Nat'l Cheng Kung, Tainan, Taiwan, 3Internal Medicine, Medical Coll/Nat'l Cheng Kung, Tainan, Taiwan, 4Biochemistry and Molecular Biology, Medical Coll/Nat'l Cheng Kung, Tainan, Taiwan, 5Microbiology and Immunology, College of Medicine, National Cheng Kung University, Medical Coll/Nat'l Cheng Kung, Tainan, Taiwan

    Background/Purpose: Synovial fibroblasts (SF) with aberrant expression of microRNAs (miRNA) are critical pathogenic regulators of rheumatoid joint, and studies examining the effect of overexpressing or…
  • Abstract Number: 1728 • 2013 ACR/ARHP Annual Meeting

    Amelioration Of Collagen-Induced Arthritis By Modulation Of Inhibitory Apoptosis Stimulating Protein Of p53 To Activate Transcription Factor p73

    Chrong-Reen Wang1, Shih-Yao Chen2, Ai-Li Shiau3, Yuan-Tsung Li4, Chia-Tse Weng5, I-Ming Jou6, Ming-Fei Liu7 and Chao-Liang Wu2, 1Section of Rheumatology and Immunology, Department of Internal Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan, 2Biochemistry and Molecular Biology, National Cheng Kung University Medical College, Tainan, Taiwan, 3Microbiology and Immunology, National Cheng Kung University Medical College, Tainan, Taiwan, 4Biochemistry and Molecular Biology, College of Medicine, National Cheng Kung University, Tainan, Taiwan, 5Internal Medicine, National Cheng Kung University Medical College, Tainan, Taiwan, 6Orthopedics, National Cheng Kung University Medical College, Tainan, Taiwan, 7Internal Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan

    Background/Purpose: Our previous studies have demonstrated p53 mutations competent for the inactivation of wild type p53 in synovial fibroblasts (SF) from either rheumatoid arthritis patients…
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