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Session » (0098–0114) Spondyloarthritis Including Psoriatic Arthritis – Basic Science Poster

Date: Sunday, October 26, 2025

Time: 10:30AM-12:30PM

Meeting: ACR Convergence 2025

10:30AM-12:30PM
Abstract Number: 0114
A rare variant in the Extl3 gene is associated with altered Wnt signaling pathway in a novel mouse model of axial spondyloarthritis
10:30AM-12:30PM
Abstract Number: 0109
Application of Psoriatic Arthritis Mouse Models in Preclinical Pharmacodynamic Evaluation
10:30AM-12:30PM
Abstract Number: 0103
Common and Rare Variant Contributions to Familial Aggregation in Spondyloarthritis
10:30AM-12:30PM
Abstract Number: 0104
Dissecting the Genetic and Functional Association of CARD9 with Axial Spondyloarthritis
10:30AM-12:30PM
Abstract Number: 0101
Enrichment of putative bacteria-reactive gut-derived IL-17+ tissue resident memory helper T cells in arthritic ankles in the SKG mouse model of spondyloarthritis
10:30AM-12:30PM
Abstract Number: 0108
In Vivo and In Vitro Analysis of IL-23 Modulation Following Anti-TNF Therapy in Psoriatic Arthritis
10:30AM-12:30PM
Abstract Number: 0105
Inflammatory Cytokines, Matrix Metalloproteinases and Bone Markers Expressions Are Modulated in the Joints in the Chronic Murine Model of Imiquimod-Induced Psoriasis
10:30AM-12:30PM
Abstract Number: 0106
Investigation of DNA Methylation Inhibition in a Mouse Model of Ankylosing Spondylitis
10:30AM-12:30PM
Abstract Number: 0102
Iron Metabolism Dysregulation and Inflammation in Ankylosing Spondylitis: Role of SLC39A14 in Extracellular Matrix Remodeling
10:30AM-12:30PM
Abstract Number: 0111
Predicting Response To Adalimumab In Patients With Psoriatic Arthritis Using Epigenetic Chromosome Conformation Signatures
10:30AM-12:30PM
Abstract Number: 0112
Proteome-wide Mendelian Randomization Identifies Therapeutic Targets in Psoriatic Arthritis
10:30AM-12:30PM
Abstract Number: 0107
Role of Achilles Elastography in Differentiating Patients with Early Psoriatic Arthritis
10:30AM-12:30PM
Abstract Number: 0110
SKG Mice Develop CD4⁺ T Cell–Driven Psoriasis and Enable Study of Endogenous Antigen-Specific Responses
10:30AM-12:30PM
Abstract Number: 0098
The Deubiquitinase TRABID is a Regulator of Osteogenesis and Inflammation in Spondyloarthritis:
10:30AM-12:30PM
Abstract Number: 0100
Therapeutic Modulation of NAD+ Metabolism in Inflammatory Rheumatic Disorders by TNFi and NAD+ Precursors
10:30AM-12:30PM
Abstract Number: 0099
Time course and impact of  IL17A on hepatic inflammation and fibrosis in adjuvant induced arthritis
10:30AM-12:30PM
Abstract Number: 0113
ZEB2 and MLCK1 may impair gut barrier integrity in MIF-Deficient SKG Mice

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Embargo Policy

All abstracts accepted to ACR Convergence are under media embargo once the ACR has notified presenters of their abstract’s acceptance. They may be presented at other meetings or published as manuscripts after this time but should not be discussed in non-scholarly venues or outlets. The following embargo policies are strictly enforced by the ACR.

Accepted abstracts are made available to the public online in advance of the meeting and are published in a special online supplement of our scientific journal, Arthritis & Rheumatology. Information contained in those abstracts may not be released until the abstracts appear online. In an exception to the media embargo, academic institutions, private organizations, and companies with products whose value may be influenced by information contained in an abstract may issue a press release to coincide with the availability of an ACR abstract on the ACR website. However, the ACR continues to require that information that goes beyond that contained in the abstract (e.g., discussion of the abstract done as part of editorial news coverage) is under media embargo until 10:00 AM CT on October 25. Journalists with access to embargoed information cannot release articles or editorial news coverage before this time. Editorial news coverage is considered original articles/videos developed by employed journalists to report facts, commentary, and subject matter expert quotes in a narrative form using a variety of sources (e.g., research, announcements, press releases, events, etc.).

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