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ACR Convergence 2026

November 6-11, 2026. Orlando, Florida.

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  • Abstract Number: 0001

    A Single-Cell and Spatial Transcriptomic Atlas of the Inflamed Aorta: Decoding the Pathogenesis of Clinically Isolated Aortitis
  • Abstract Number: 0002

    Age-Related Antigen Discovery in Giant Cell Arteritis
  • Abstract Number: 0003

    Association of Increasing Genetically-Mediated Complement Activity with ANCA-Associated Vasculitis
  • Abstract Number: 0004

    Baseline and early longitudinal serum biomarkers in Polymyalgia Rheumatica: kinetics and associations with clinical course
  • Abstract Number: 0005

    Is the HLA-B*52 allele associated with an increased risk of developing Takayasu arteritis in Brazil? A Monocenter Cross-Sectional Study.
  • Abstract Number: 0006

    Polygenic Risk for Nasal Polyps in ANCA-Associated Vasculitis
  • Abstract Number: 0008

    Transcriptome analysis of temporal artery and aortic lesions highlights vessel-specific chemotaxis and cell adhesion in GCA
  • Abstract Number: 0009

    An Ex-Vivo Functional Immune Assay Predicts TNF Inhibitor Response Across T Cell-Mediated Autoimmune Diseases
  • Abstract Number: 0010

    Induction of B Cell Aplasia with cGP130 Armored-CAR T Cells Without Lymphodepletion
  • Abstract Number: 0011

    PD-1+CD8+ T cells are hyperactivated and associated with refractory systemic lupus erythematosus
  • Abstract Number: 0012

    Peripheral T Cell Immunophenotyping Identifies Terminally Differentiated Cytotoxic CD8 T Cell Expansion, Effector Memory T Cell Depletion, and anti-Ro-Associated CD38⁺ CD4 T Cell Activation in Sjögren’s Disease
  • Abstract Number: 0013

    Profiling U170-Specific Self-Reactive CD4+ T cells in MCTD and SLE
  • Abstract Number: 0014

    PsoriAxis Reveals Continuous Molecular Healing and Residual Disease States in Psoriasis
  • Abstract Number: 0015

    Ro+ Sjögren’s disease patients display selective enrichment of TCR V/J genes in the salivary gland with a distinguishing clonal profile
  • Abstract Number: 0016

    Small-Molecule Inhibitors of TNF Reduce Pro-inflammatory Disease Activity while Retaining TNFR2 Signaling, Which Preserves the Suppressive Capacity of Regulatory T Cells and Promotes Immune Tolerance
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